Anyone can experience temporary paresthesia. Your risk of radiculopathy increases with age. You also may be more prone to it if you:. Be prepared to give your medical history. Mention any activities you participate in that involve repetitive movement. You should also list any over-the-counter or prescription medications that you take. Your doctor will consider your known health conditions to help them make a diagnosis. If you have diabetes, for example, your doctor will want to determine if you have nerve damage, or neuropathy.
Your doctor will probably perform a full physical exam. This will likely include a neurological exam as well. Blood work and other laboratory tests, such as a spinal tap, may help them rule out certain diseases. Depending on the results, they may refer you to a specialist, such as a neurologist, orthopedist, or endocrinologist.
Treatment depends on the cause of your paresthesia. It may be possible to treat your condition by eliminating the cause in some cases. For example, if you have a repetitive movement injury, a few lifestyle adjustments or physical therapy may solve the problem.
If your paresthesia is due to an underlying disease, getting treatment for that disease can potentially ease the symptoms of paresthesia. Your individual circumstances will determine whether your symptoms will improve. Some types of nerve damage are irreversible. It can complicate your daily life if the symptoms are severe. The severity of chronic paresthesia and how long it will last largely depends on the cause. In some cases, treating the underlying condition solves the problem.
Having made the diagnosis, many of the conditions, but particularly carpal tunnel syndrome and ulnar neuropathy, can be effectively managed by the GP without requiring consultant involvement. Addressing the underlying problem is often all that is required but, in complex cases, referral to a specialist may be necessary.
Competing interests: None. Provenance and peer review: Commissioned, externally peer reviewed. Australian Family Physician. Search for: Search AFP. Filter Relevance Date. Issues by year. Volume 44, Issue 3, March Background Paraesthesia reflects an abnormality affecting the sensory pathways anywhere between the peripheral sensory nervous system and the sensory cortex. As with all neurology, the fundamental diagnostic tool is a concise history, devoid of potentially ambigu-ous jargon, which properly reflects the true nature of what the patient is experiencing, provocateurs, precipitating and relieving factors, concomitant illnesses, such as diabetes, and any treatments that could evoke neuropathies.
Nerve conduction studies may be necessary to diagnose peripheral neuropathy, but they may also be normal with small fibre neuropathy. Even with a diagnosis of peripheral neuropathy, definition of the underlying cause may remain elusive in a significant proportion of cases, despite involvement of consultants. Discussion Treatment is based on the relevant diagnosis and mechanism to address the cause.
This includes better glycaemic control for diabetes, night splint for CTS or elbow padding for ulnar neuropathy, modifying lifestyle with reduced alcohol consumption or replacing dietary deficiencies or changing medications where appropriate and practical. Should such intervention fail to relieve symptoms, consideration of intervention to relieve symptoms of neuropathic pain may be required. History As is the case with all neurology, the most important diagnostic tool is a detailed comprehensive history that reflects what the patient is trying to convey.
Table 1. Investigations Neurophysiology, using nerve conduction studies and electromyography, may be required, especially for the patient who proves to be a very poor historian or in whom confirmation of a diagnosis may dictate altered therapy.
Treatment Treatment is determined by the relevant diagnosis. Conclusion As with all neurological conditions, the most important diagnostic tool is a good history, complemented by physical examination — usually to confirm the diagnosis, already considered, based on the history obtained. References Beran RG. Sydney: Elsevier, Search PubMed Curtis L.
Nutritional approaches to treat diabetic neuropathy: a systematic review. Int J Diab Research ;— Parkinsonism Relat Disord ;— Chemotherapy-induced peripheral neuropathy in adults: A comprehensive update of the literature. Objective: Some localised neuropathies, such as carpal tunnel syndrome CTS or ulnar neuropathy, produce classical features, such as weakness of the 'LOAF' lateral two lumbricals, opponens pollicis, abductor pollicis brevis and flexor pollicis brevis median innervated muscles, thereby obviating need for further neurophysiology.
Nerve conduction studies may be necessary to diagnose peripheral neuropathy, but they may also be normal with small fibre neuropathy. Neuropathy can result in muscle weakness if it causes reduced nervous stimulation to the muscles. This can occur in any muscle including chest muscles making breathing difficult or in the lower extremities resulting in atrophy and difficulty walking. There are many causes of peripheral neuropathy 1.
It is a fairly common symptom of Th1 diseases The chronic inflammatory diseases caused by bacterial pathogens. It is a protective attempt by the organism to remove the injurious stimuli as well as initiate the healing process for the tissue.
Irritation to the nerve can also come from inflammation to the surrounding tissue such as Rheumatoid Arthritis. Impaired function and symptoms depend on the type of nerves — motor, sensory, or autonomic — that are damaged. Some people may experience temporary numbness, tingling, and pricking sensations, sensitivity to touch, or muscle weakness. Others may suffer more extreme symptoms, including burning pain especially at night , muscle wasting, paralysis, or organ or gland dysfunction.
Resolving neuropathy and paresthesia involves identifying and eliminating the underlying cause. The Marshall Protcol is designed to treat the underlying cause of Th1 inflammation The complex biological response of vascular tissues to harmful stimuli such as pathogens or damaged cells. Many MPers report resolution of their paresthesia.
0コメント